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They are often discussed together because both are associated with recovery and tissue-support pathways
The published data show that estrogen-like molecules can mimic or block endogenous estrogens biological effects through a series of non-genomic responses (Park et al., 2012
Oxidative stress related to iron overload markedly increases malondialdehyde, a marker of lipid peroxidation [85], and high levels of myeloperoxidase released by activated leukocytes and neutrophils, causing endothelial dysfunction and liver injury [86]
[DOI] [PubMed] [Google Scholar] 127.Gomes M
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