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Polyubiquitination of transforming growth factor -activated kinase 1 (TAK1) at lysine 562 residue regulates TLR4-mediated JNK and p38 MAPK activation

The increased satiating potency of CCK-8 by estradiol is not mediated by upregulation of NTS CCK receptors

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ROS has long been recognized as a harmful substance, and its excessive accumulation will cause the release of inflammatory factors, enhance the adhesion of inflammatory factors to endothelial cells, and promote apoptosis and even cause cell death (Forrester et al., 2018)

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