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Such genetic variations may restrict the efficiency of selenium transport, consequently diminishing the activity of selenium-dependent enzymes
This is where the real question comes in
TRPA1 and TRPV4 mediate paclitaxel-induced peripheral neuropathy in mice via a glutathione-sensitive mechanism
[DOI] [Google Scholar] Ling XC, Kuo KL
However, numerous studies have shown that, aside from its anti-protease properties, AAT has anti-inflammatory and immune regulatory functions, and some of these functions seem to be independent of the anti-protease activities
