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Increased flux of key cellular energy regulators, including NAD + and SAM, may potentially define the therapeutic mechanism-of-action of NNMT inhibitors

A rational design of therapeutic combination strategies is needed to flatten cellular homeostasis and accomplish a drop in cancer development

(Springer, Cham: Springer Nature) 33745

In these disorders, increased OS can occur due to the exhaustion of antioxidants, mitochondrial dysfunction, glutamatergic excitotoxicity, neuro-inflammation, raised expression of pro-apoptotic proteins, and possible genetic modifications [28]