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Key Points Glucocorticoids modulate growth hormone (GH) secretion at both the hypothalamic and pituitary level Hypoadrenalism might cause GH deficiency that is reversible during glucocorticoid replacement therapy Testing for GH deficiency in patients with hypopituitarism should always be performed after adequate periods of cortisol replacement Chronic excess of glucocorticoids, either exogenous or endogenous, causes GH deficiency that might be involved in metabolic and cardiovascular complications Treatment with recombinant GH could be considered in children and adults with glucocorticoid-induced GH deficiency This is a preview of subscription content, access via your institution Access options Subscribe to this journal Receive 12 print issues and online access 186,36 per year only 15,53 per issue Buy this article Purchase on SpringerLink Instant access to the full article PDF

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furthermore, the in vitro migration and rate of spreading of tendon fibroblasts increased in a dose-dependent manner, which was attributed to the activation of the FAK-paxillin pathway

The biosynthesis of coenzyme Q 10 involves three major steps: (1) synthesis of the benzoquinone structure from 4-hydroxybenzoate derived from either tyrosine or phenylalanine, two amino acids