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However, the hypercoagulable state, as well as the molecular mechanism of this aspect of the pathogenesis of -thalassemia/HbE, remains poorly understood

Some of them are presented illustratively in the table below (Tab.1) Background diseases Disease name Causes Symptoms Treatment Bacterial Impetigo contagiosa Strains of staphylococci or streptococci Blistering wounds, most often around the mouth and nose Antibiotic therapy BPC-157 peptide therapy Fungal Nail fungus Infection with pathogenic fungi Discoloration, thickening, ridging, nail deformation Topical treatment Oral treatment BPC-157 peptide therapy Allergic Atopic dermatitis Genetic, environmental, or immunological Itching, dryness, and skin discoloration Oral treatment including antihistamines Topical treatment BPC-157 peptide therapy Diseases connective tissue Systemic lupus erythematosus Genetic predispositions Erythema, ulcers, and skin inflammations Slowing disease progression through: Oral therapy Therapy with the peptide BPC-157 Table 1

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