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Pharmacological targeting of kinases MST1 and MST2 augments tissue repair and regeneration

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Gene and cell therapies represent another promising avenue, aiming to treat the underlying causes of ophthalmic diseases rather than merely managing symptoms

One study showed that ROS-mediated modification of residues of cysteine 257, 268, and 586 can lead to the formation of an intramolecular disulfide bond on RIP1, which promotes the autophosphorylation of RIP1 at serine 161 thereby effectively recruiting RIP3