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GPX4 at the crossroads of lipid homeostasis and ferroptosis

While the precise mechanism for the disruption of the ligand-binding interaction between these two proteins is not defined, the presence of an aggregation of GST monomers following oxidative stress implicates intermolecular disulfide bridge formation between available cysteine residues

Our IHPC treatment pattern was as follows: mice were treated with 10 cycles of intermittent hypoxia per day

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