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doi: 10.1016/j.mrfmmm.2009.05.010 17 LowryMHMcAllisterBPJeanJCBrownLAHugheyRPCruikshankWWet al

Under oxidative stress, excessive ROS such as OH directly attack DNA, causing single-strand or double-strand breaks, activating PARP-1, consuming NAD and ATP, and generating poly(ADP-ribose), resulting in mitochondrial membrane potential loss and the release of apoptosis-inducing factor, ultimately resulting in cell death [252]

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Moreover, quercetin-treated rats exhibited improved social behavior and learning, which were severely impaired in the PPAS group