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Inhibition of TrxR leads to increased ROS levels in cancer cells to a lethal level, consequently leading to activation of cell death mechanisms

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In the setting of high ROS, as is seen with G6PD-inhibition, heme production has been shown to be impaired 137

Notably, the apparent reduction of p-IGF1R in heterotopic tumors from IGF1R deficient mice was reflected in reduced p-IGF1R protein levels measured by ELISA (Fig

This imbalance in Treg:Teff functions in highly glycolytic tumors likely contributes to their noteworthy resistance to ICB and TIL therapy 199, 202, 203