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Future studies are needed to reconcile the underlying differences between tumor tissue and basal PTC cell line GPX4 expression levels and in vitro responses to pharmacologic inhibition of GPX4 within an emulated 3D thyroid tumor microenvironment in vitro and in vivo
SETDB1 is responsible for the methylation of histone H3 on lysine 9 (H3K9) and is recurrently amplified in melanoma to play an oncogenic role
