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Both acute and chronic kidney conditions such as those caused by toxins, ischemia, or inflammation affect the renal tubules, leading to renal fibrosis and a progressive decline in GFR (Mackensen-Haen et al

Both priming and activation are blocked by treatment with genistein, a tyrosine kinase inhibitor, suggesting an important role for tyrosine kinase signaling pathway in activating NADPH oxidase (McLeish et al., 1998

Next, we evaluated accumulation of intracellular lipid peroxides, a hallmark of ferroptosis, using flow cytometry

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Quantitative analysis of atherosclerotic lesions, plaque areas, and positively stained areas was conducted using ImageJ