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Abstract Cerebral ischemia, a leading cause of neurological disability and death, is characterized by reduced cerebral blood flow that induces hypoxia, neuronal injury, and irreversible brain damage

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AP1 is a complex of several proteins including JUN, which is upregulated in motor neurons derived from SOD1 patients compared to other neurons, providing potential mechanistic insights into the selective degeneration of motor neurons in ALS (Bhinge et al., 2005)

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MDSUPDRS motor scores and data on dopaminergic treatment were not available for the subjects covered for the transcriptomics profiling, and therefore the comparison of metabolomics and transcriptomics data focused on the case-control analyses and shared network alterations, confirming the treatment-independence using the metabolomics data (see section on Pathway and network analyses below)