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The alleviation of THO, as well as the depletion of NK/NKT cells, significantly reduced the presence of activated DCs in the inflamed eyes of DED mice, alleviated their antigen-presenting properties, attenuated Th1 and Th17 cell-driven eye inflammation, down-regulated MMP-3 and MMP-9 expression, and reduced apoptosis of their corneal and conjunctival epithelial cells, confirming the important pathogenic role of THO in NK and NKT cell-driven inflammation in the eyes of DED patients [58]

Clinical trial design in ALS continues to advance, allowing for better measurement of changes in outcomes via patient stratification, identification of patients most likely to benefit from specific drug mechanisms, and methods for faster drug development and availability to patients

doi: 10.1016/j.archoralbio.2016.07.004

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