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and the interplay between the endoplasmic reticulum and actin to drive the contraction of the Drp1 protein to reduce the molecular spacing, leading to the rupture of the OMM and IMM and ultimately triggering mitochondrial fission (Giacomello et al., 2020

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The potential for N-Acetyl Semax Amidate to mirror these effects remains a subject for future research

Knox JJ, 2009, 'Rationale and evidence for sunitinib in the treatment of malignant paraganglioma/pheochromocytoma', Journal of Clinical Endocrinology and Metabolism , 94, pp

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