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In SLE, these mechanisms restore immune balance by curbing pathogenic Th2 overactivation (e.g., IL-5/IL-13) while enhancing regulatory IgA synthesis and Treg-mediated suppression of autoreactive B cells (51, 129)

It is administered as an injection under medical supervision and should not be self-injected unless trained by a healthcare professional

Materials and methods For biological analysis, MSC and HUVEC viability after freeze-thawing and alteration of gene expression in response to dimethyl sulfoxide (DMSO, a cryoprotectant) were quantitatively evaluated

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In atherosclerosis, NAC therapy appeared to inhibit extracellular degradation and to improve vascular stability in both the early and late stages of atherosclerosis