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Description
Replacing glutathione or having GSH treatment, therefore, would seem to make logical sense
Protective properties of GSB-106 arise from its ability to counteract cell apoptosis via activation of TrkB-dependent pro-survival mechanisms, including inactivation of pro-apoptotic BAD protein and suppression of caspases 9 and 3/7
The central compartment consists of the plasma, extracellular fluids and highly perfused tissues in which drug equilibrate rapidly
Antioxidant treatment of ischemic brain lesions
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