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Direct activation of GABAA receptors by barbiturates in cultured rat hippocampal neurons

Nilotinib is a tyrosine kinase c-Abl inhibitor approved for the treatment of leukemia, that has also shown to increase autophagy and degradation of intracellular -syn aggregates (Fig

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found that significant enrichment of Th2 and Th17 lymphocyte subsets identified within bronchoalveolar lavage samples obtained from individuals diagnosed with asthma could trigger IL-4 overproduction and subsequently augment the capability of inducing glucocorticoid resistance in vitro [131]