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Viscarra JA, Vzquez-Medina JP, Crocker DE, Ortiz RM

This cascade leads to neuronal apoptosis and energy deficits, particularly in the prefrontal cortex and hippocampus ( 4.3 Excitotoxicity Synthetic cathinones disrupt glutamatergic signaling by enhancing glutamate release and impairing astrocytic uptake ( 4.4 Neuroinflammation The neurotoxic environment created by synthetic cathinones activates microglia and astrocytes, leading to the release of proinflammatory cytokines, such as IL-1 and TNF-

Hermann et al., 2000

it also helps recycle other antioxidants, keeping them active for longer

Its ability to target multiple pathways involved in type 2 inflammation and oxidative stress makes it a compelling candidate for further exploration