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Metabolites in the transmethylation and transsulfuration pathways, responsible for glutathione production are imbalanced in those with this disorder

C1q + tumor-associated macrophages contribute to immunosuppression through fatty acid metabolic reprogramming in malignant pleural effusion

The exploration of these sulfur-containing amino acids opens avenues for understanding pathophysiological mechanisms and suggests potential targets for therapeutic interventions

The transcriptional coactivators, pgc-1 and , cooperate to maintain cardiac mitochondrial function during the early stages of insulin resistance