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Various medications used to treat ulcerative colitis will target specific parts of the immune system, stopping the inflammatory response responsible for UC symptoms

The TrxRs differ from GR by having a catalytic selenocysteine redox-active site at the C-terminus in addition to the standard CXXC domain (Lu and Holmgren, 2014)

Lipoplexes, for instance, have been shown to fuse with endosomal membranes, and cationic polymers such as PEI and fusogenic lipids such as cholesterol and DOPE facilitate endosomal release [7]

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Ferringer, MD, FAAD, Chair Khalid Ali Al Hawsawi, MD Neal D