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Abnormally elevated levels of DRP1 not only cause mitochondrial fission, mitochondrial fragmentation, and decreased efficiency of mitochondrial ATP production but also impede glucose uptake (104) and glucose-stimulated insulin secretion by inhibiting the translocation of the insulin receptor substrate 1-protein kinase B (IRS1-Akt) pathway and glucose transport proteins in mouse pancreatic -cells (105)

Accordingly, antioxidant pharmacotherapies have been applied to explore efficacious strategies for the protection of neural cells suffering from oxidative stress generated by METH

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80 Furthermore, mitochondria in immune cells from fibromyalgia patients were found to have structural abnormalities and the number of affected mitochondria corresponded with severity of pain