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Blood-brain barrier transport machineries and targeted therapy of brain diseases

Therefore, we treated in vitro activated B6 FoB with various mitochondrial inhibitors and the Gclc inhibitor BSO and measured total immunoglobulins from the supernatants

doi: 10.1016/j.biomaterials.2017.12.025 71 AiTShangLLiBLiJQinR

This is the theoretical basis for endogenously supplied gamma-glutamylcysteines ability to raise GSH levels, as being the product of the GCL enzyme, it can be taken up by cells intact, where it feeds directly into glutathione synthase to be converted to glutathione, effectively bypassing the dysfunctional GCL

doi: 10.1016/j.jtemb.2005.04.005