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Figure 2 Pathological Effect Caused by PDH Imbalance As a critical node in metabolic regulation, PDHC converts pyruvate to acetyl-CoA in mitochondria, and its inhibition leads to early metabolic reprogramming in response to sepsis, resulting in the accumulation of lactic acid, the aggravation of inflammation, the remodeling of TCA cycle, and the disorder of lipid metabolism, and the consequence of lower ATP synthesis relative to ATP demand

Many tumors develop a metabolic dependency due to the lack of a working methionine salvage pathway, which can be targeted by methionine restriction (MR)

Magnesium glycinate is highly bioavailable and gentle on the stomach, making it ideal for long-term neurological and muscular support

doi: 10.1007/s11695-017-2833-0