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[DOI] [PMC free article] [PubMed] [Google Scholar] 840.Burns R.S., Chiueh C.C., Markey S.P., Ebert M.H., Jacobowitz D.M., Kopin I.J

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In cultured cells, GLP-1 mediates its effects through G proteins linked to adenylyl cyclase, resulting in increased intracellular circulating adenosine monophosphate and activation of protein kinase A (PKA) [17]

The breakdown shows 14.3% of patients have minimal retention, 44.3% show moderate amounts, and 41.4% have large quantities [3]

Each agent differs in pharmacokinetic properties, therapeutic efficacy, adverse event profiles, and modes of administration, necessitating individualized evaluation.84 Clinicians must be well informed about the advantages and limitations of each GLP-1RAs to guide optimal therapeutic decision-making